Vitamin E (Vitamin E) yog ib pawg ntawm fat-soluble vitamins uas muaj α, β, γ, δ-tocopherol thiab tocotrienol. Nws yog ib qho tseem ceeb uas lub cev tsis tuaj yeem tsim los ntawm nws tus kheej, thiab yog ib qho tseem ceeb tshaj plaws antioxidants 12.
Cov qauv tshuaj muaj xws li benzopyrane rings thiab hydrophobic sab chains, uas muab cov lipid membrane nkag mus rau 67.
lub cev thiab tshuaj muaj zog

Solubility: soluble nyob rau hauv roj, ethanol thiab lwm yam organic solvents, insoluble nyob rau hauv dej 12.
Kev ruaj ntseg: kub resistant (≤200 ° C) thiab acidic ib puag ncig tab sis rhiab rau alkali, oxygen, UV rays thiab hlau ions (Fe3 + + / Cu2 +). Frying significantly rhuav tshem cov haujlwm

Ua ntej, antioxidant synergistic mechanism dawb radical scavenging circulatory system

Vitamin E, raws li ib tug fat-soluble antioxidant, nyiam neutralizes lipid dawb radicals nyob rau hauv cell membranes, thaum vitamin C, raws li ib tug dej-soluble antioxidant, txo thiab regenerates oxidized vitamin E (tocopherol dawb radicals), tsim ib tug nruam antioxidant voj voog 17.
Cov kev sim qhia tau hais tias kev sib xyaw ua ke ntawm ob tuaj yeem txhim kho cov tshuaj tiv thaiv antioxidant los ntawm 3 zaug, ib qho molecule vitamin E tuaj yeem tshem tawm mus txog 4 dawb radicals, vitamin C los ntawm cov txheej txheem tsim kho kom ntev nws lub sijhawm ua haujlwm 78.

Interphase Defense network

Vitamin E anchors lub lipid bilayer ntawm cell membrane thiab thaiv cov lipid peroxidation saw cov tshuaj tiv thaiv. Vitamin C scavenge dej-soluble dawb radicals nyob rau hauv cytoplasmic matrix los tsim ib lipid - dej biphasic tiv thaiv system 13.

Ob, lub cev tiv thaiv kab mob bidirectional ua kom cov nyhuv ua kom lub cev tiv thaiv kab mob: Vitamin C txhawb nqa neutrophil chemotaxis, vitamin E txhim kho NK cell kev ua, thiab kev sib xyaw ua ke txo qhov tshwm sim ntawm tus kab mob ua pa los ntawm 32% 15.
tswj hwm kev tiv thaiv kab mob: kev sib koom ua ke txhawb T lymphocyte proliferation, txhim kho cov tshuaj tiv thaiv kev ua haujlwm ntawm 28%, thiab muaj txiaj ntsig zoo ntawm cov tshuaj tiv thaiv 57.

Thib peb, kev noj qab haus huv ntawm daim tawv nqaij sib koom ua ke txoj hauv kev

Kev tiv thaiv kev yees duab

Vitamin E inhibits UV-induced sepiperoxidation thiab vitamin C blocks tyrosinase kev ua, ib qho tseem ceeb enzyme nyob rau hauv melanin synthesis, thiab kev sib xyaw ua ke txo tus nqi ntawm daim tawv nqaij erythematosis los ntawm 54% ??24.

Kev tswj hwm ntawm collagen metabolism

Vitamin C activates prolyl hydroxylase los txhawb collagen synthesis, thiab vitamin E txo cov kev ua ntawm collagenase los tiv thaiv degradation. Cov kev sim tshuaj ntsuam xyuas tau pom tias daim tawv nqaij elasticity tau txhim kho los ntawm 23% thiab qhov tob tob yog txo los ntawm 19% 34.

4. Kev sib koom ua ke ntawm kev tiv thaiv kab mob plawv
Kev cuam tshuam atherosclerosis : Vitamin E inhibits oxidation ntawm low-density lipoprotein (LDL) thiab vitamin C kho vascular endothelium ua ke nrog 18% qis dua ntawm cov xwm txheej hauv plawv 15.
Kev txhim kho microcirculation : vitamin E txo qis platelet sib sau ua ke, vitamin C txhim kho capillary toughness, thiab muaj txiaj ntsig ntawm kev sib koom tes tiv thaiv thiab kho mob ntshav qab zib retinopathy 58.

5. Metabolic kev koom tes optimization yam ntxwv
Kev tswj cov hlau metabolism: Vitamin C yuav txo cov hlau trivalent rau bivalent hlau, txhim kho qhov nqus ntawm 2-3 zaug; Vitamin E tiv thaiv erythrocyte membrane stability thiab txo qhov kev pheej hmoo ntawm hemolysis 57.
Cov roj cholesterol tshuav : Vitamin E inhibits HMG-CoA reductase kom txo cov roj cholesterol synthesis, vitamin C txhawb cov kua tsib kua qaub tawm, thiab ua ke txo cov roj cholesterol tag nrho los ntawm 12% -15%.