Vitamin E (Vitamin E) b? otu vitamin na-agbaze ab?ba nwere α, β, γ, δ-tocopherol na tocotrienol. ? b? ihe d? mkpa na-edozi ah? nke ah? enwegh? ike ?mep?ta onwe ya, ? b?kwa otu n'ime ihe nd? kas? mkpa antioxidants?12.
?d?d? kem?kal? nwere mgbanaka benzopyrane na agb? ak?k? hydrophobic, nke na-enye ntinye nke akp?kp? ah? lipid 67.
Njirimara an? ah? na kem?kal

Solubility : soluble na ab?ba, ethanol na nd? ?z? organic solvents, insoluble na mmiri 12.
Nkw?si ike : okpom?k? na-eguzogide (≤200 Celsius C) na gburugburu acidic ma na-enwe mmet?ta maka alkali, oxygen, UV rays na ion metal (Fe3 + + /Cu2 +). Ighe na-emebi ?r? ah? nke ukwuu

Nke mb?, sistemu synergistic antioxidant sistemu ?bara na-ekpochap? radical free

Vitamin E, d? ka ab?ba soluble antioxidant, ?kachamma neutralizes lipid free radicals na cell membranes, mgbe vitamin C, d? ka a mmiri-soluble antioxidant, na-ebelata na regenerates oxidized vitamin E (tocopherol free radicals), na-akp? a na-aga n'ihu antioxidant okirikiri ?17.
Nnwale na-egosi na nchikota nke ab?? nwere ike imeziwanye ar?m?r? antioxidant site ugboro 3, otu molekul vitamin E nwere ike na-ewep? ihe ruru 4 free radicals, vitamin C site na usoro mmeghari iji mee ka oge ya d? ogologo ?78.

Interphase Defence netw?k

Vitamin E na-etinye eriri lipid bilayer nke akp?kp? ah? cell ma na-egbochi mmeghachi omume lipid peroxidation chain. Vitamin C na-ekpochap? radicals n'efu na mmiri na-agbaze na cytoplasmic matrix iji wuo lipid-water biphasic protection system 13.

Ab??, usoro ah? ji al?so ?r?a ?g? bidirectional activation mmet?ta na-eme ka ihe mgbochi ebum p?ta: Vitamin C na-akwalite chemotaxis neutrophil, vitamin E na-eme ka ?r? sel NK d?kwuo mma, na nchikota na-ebelata ohere nke ?r?a iku ume site na 32%?15.
Na-ach?kwa mgbochi mgbanwe : synergistically na-akpalite mm?ba nke T lymphocyte, na-eme ka mmep?ta mmep?ta ihe mgbochi site na 28%, ma nwee mmet?ta bara uru na nzaghachi ?gw? mgbochi 57.

Nke at?, ?z? enyemaka nkwonkwo ah?ike an? ah?

Nchekwa foto

Vitamin E na-egbochi sepiperoxidation nke UV na-ebute na vitamin C na-egbochi ?r? tyrosinase, enzyme b? isi na njik? melanin, na ngwak?ta na-ebelata ?n? ?g?g? nke erythematosis akp?kp? site 54%?24.

Usoro nke metabolism nke collagen

Vitamin C na-ar? ?r? prolyl hydroxylase iji kwalite njik? nke collagen, na vitamin E na-ebelata ?r? collagenase iji gbochie mmebi. Nnwale ?l??gw? egosila na a na-eme ka nh?ah? akp?kp? ah? d?kwuo mma site na 23% na omimi nke wrinkle belatara site na 19%?34.

4. Usoro imek? ihe ?n? nke nchebe obi
Ntinye aka nke atherosclerosis: Vitamin E na-egbochi oxidation nke obere njup?ta lipoprotein (LDL) na vitamin C na-edozi endothelium vask?la mebiri emebi yana 18% obere ihe ize nd? nke ihe omume obi ?15.
Mm?ba microcirculation: vitamin E na-ebelata nch?k?ta platelet na-ad?gh? mma, vitamin C na-eme ka ike sie ike, ma nwee uru nke mgbochi nkwonkwo na ?gw?gw? nke retinopathy na-ar?a ?r?a shuga 58.

5. Metabolic nkwado njikar?cha njirimara
Iron metabolism regulation : Vitamin C ga-ebelata trivalent ígwè na bivalent ígwè, melite absorption ?n?ego 2-3 ugboro; Vitamin E na-echebe nkw?si ike nke akp?kp? ah? erythrocyte ma na-ebelata ihe ize nd? nke hemolysis ?57.
Nt?le cholesterol: Vitamin E na-egbochi HMG-CoA reductase iji belata njik? cholesterol, vitamin C na-akwalite excretion nke bile acid, yana ngwak?ta na-ebelata mkpok?ta cholesterol site na 12% -15%.